Delayed-onset parkinsonism after carbon monoxide poisoning: neuropsychological perspectives from a case report.
Delayed-onset parkinsonism after carbon monoxide poisoning: neuropsychological perspectives from a case report.
Where did the research take place?
The study site has not been established. Author addresses may differ from where the research occurred.
Brescia, IT · Author affiliation
Neurology Operating Unit, Fondazione Poliambulanza Istituto Ospedaliero, Brescia, Italy.Location evidence
Cattolica, IT · Author affiliation
Department of Psychology, Università Cattolica Del Sacro Cuore, Milan, Italy.Location evidence
Milan, IT · Author affiliation
Department of Psychology, Università Cattolica Del Sacro Cuore, Milan, Italy.Location evidence
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Original abstract
This case report presents the clinical evolution of a patient who developed delayed-onset parkinsonism and cognitive deficits following carbon monoxide poisoning (COP). After a symptom-free interval of approximately one month, the patient exhibited progressive neurological and cognitive impairments that significantly interfered with daily and occupational functioning. A combined therapeutic approach was implemented, involving pharmacological treatment, cycles of hyperbaric oxygen therapy (HBOT) and a tailored neuropsychological rehabilitation program. Longitudinal assessments documented progressive improvements, particularly in sustained attention, memory, and executive functioning, although residual difficulties persisted. This case highlights the relevance of individualized neuropsychological interventions in conjunction with medical treatment in facilitating functional recovery in patients with delayed neurological sequelae after COP.